Max in WT synaptoneurosomes, suggesting that Src signaling may very well be downregulated in KI synapses. Conversely, our power to rescue SERT functionality in KI midbrain synaptoneurosomes because of the inhibition of FAK suggests elevated FAK signaling downstream on the Pro32Pro33 mutant, as confirmed by elevated pFAK localization in 5-HT https://pro33login44433.full-design.com/pro33-login-can-be-fun-for-anyone-74785359